Enhanced Ca2+ influx through cardiac L-type Ca2+ channels maintains the systolic Ca2+ transient in early cardiac atrophy induced by mechanical unloading-Reference-Cited by-同舟云学术

Enhanced Ca2+ influx through cardiac L-type Ca2+ channels maintains the systolic Ca2+ transient in early cardiac atrophy induced by mechanical unloading

Published:2013-07-11 Issue:12 Volume:465 Page:1763-1773
ISSN:0031-6768
Container-title:Pflügers Archiv - European Journal of Physiology
language:en
Short-container-title:Pflugers Arch - Eur J Physiol

Author:

Schwoerer A. P.,Neef S.,Broichhausen I.,Jacubeit J.,Tiburcy M.,Wagner M.,Biermann D.,Didié M.,Vettel C.,Maier L. S.,Zimmermann W. H.,Carrier L.,Eschenhagen T.,Volk T.,El-Armouche A.,Ehmke H.

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Clinical Biochemistry,Physiology

Link

http://link.springer.com/article/10.1007/s00424-013-1316-y/fulltext.html

Reference55 articles.

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2. Baskin KK, Taegtmeyer H (2011) Taking pressure off the heart: the ins and outs of atrophic remodelling. Cardiovasc Res 90:243–250

3. Benitah JP, Alvarez JL, Gomez AM (2010) L-type Ca2+ current in ventricular cardiomyocytes. J Mol Cell Cardiol 48:26–36

4. Bers DM (2008) Calcium cycling and signaling in cardiac myocytes. Annu Rev Physiol 70:23–49

5. Bito V, Heinzel FR, Biesmans L, Antoons G, Sipido KR (2008) Crosstalk between L-type Ca2+ channels and the sarcoplasmic reticulum: alterations during cardiac remodelling. Cardiovasc Res 77:315–324

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