Antibodies to HLA Molecules Mimic Agonistic Stimulation to Trigger Vascular Cell Changes and Induce Allograft Injury
Author:
Publisher
Springer Science and Business Media LLC
Subject
Transplantation,Nephrology,Hepatology,Immunology,Surgery
Link
http://link.springer.com/content/pdf/10.1007/s40472-015-0065-6.pdf
Reference119 articles.
1. Frank R et al. Correlations of lymphocyte subset infiltrates with donor-specific antibodies and acute antibody-mediated rejection in endomyocardial biopsies. Cardiovasc Pathol. doi: 10.1016/j.carpath.2014.11.001
2. Torres IB et al. Comparing transplant glomerulopathy in the absence of C4d deposition and donor-specific antibodies to chronic antibody-mediated rejection. Clin Transpl. 2014;28:1148–54. doi: 10.1111/ctr.12433 .
3. DeNicola MM et al. Pathologic findings in lung allografts with anti-HLA antibodies. J Heart Lung Transplant Off Pub Int Soc Heart Trans. 2013;32:326–32. doi: 10.1016/j.healun.2012.11.018 .
4. Valenzuela NM et al. HLA class I antibodies trigger increased adherence of monocytes to endothelial cells by eliciting an increase in endothelial P-selectin, and depending on subclass, by engaging FcgammaRs. J Immunol. 2013;190:6635–50. doi: 10.4049/jimmunol.1201434 .
5. Valenzuela NM et al. Monocyte recruitment by HLA IgG-activated endothelium: the relationship between IgG subclass and FcγRIIa polymorphisms. Am J Trans. 2015. doi: 10.1111/ajt.13174 .
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