Derangement of Erythrocytic AE1 in Beta-Thalassemia by Caspase 3: Pathogenic Mechanisms and Implications in Red Blood Cell Senescence-Reference-Cited by-同舟云学术

Derangement of Erythrocytic AE1 in Beta-Thalassemia by Caspase 3: Pathogenic Mechanisms and Implications in Red Blood Cell Senescence

Published:2009-02-24 Issue:1 Volume:228 Page:43-49
ISSN:0022-2631
Container-title:Journal of Membrane Biology
language:en
Short-container-title:J Membrane Biol

Author:

Ficarra Silvana,Tellone Ester,Giardina Bruno,Scatena Roberto,Russo Annamaria,Misiti Francesco,Clementi M. Elisabetta,Colucci Deborah,Bellocco Ersilia,Laganà Giuseppina,Barreca Davide,Galtieri Antonio

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Physiology,Biophysics

Link

http://link.springer.com/content/pdf/10.1007/s00232-009-9157-5.pdf

Reference48 articles.

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2. Alekperova GA, Orudzhev AG, Javadov SA (2004) Analysis of erythrocyte and platelet membrane proteins in various forms of β-thalassemia. Biochemistry (Mosc) 69:748–753

3. Bennett V, Stenbuck PJ (1980) Association between ankyrin and the cytoplasmic domain of band 3 isolated from the human erythrocyte membrane. J Biol Chem 255:6424–6432

4. Brovelli A, Castellana MA, Minetti G, Piccinini G, Seppi C, Renzis MR, Balduini C (1991) Conformational changes and oxidation of membrane proteins in senescent human erythrocytes. In: Magnani M, De Flora A (eds) Red blood cell aging. Plenum Press, New York, p 59

5. Browne P, Shalev O, Hebbel RP (1998) The molecular pathobiology of cell membrane iron: the sickle red cell as a model. Free Radic Biol Med 24:1040–1048

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