Novel Targets in Glucose Homeostasis and Obesity—Lesson from Rare Mutations
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11892-020-01351-7.pdf
Reference87 articles.
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2. Ghosh S, Bouchard C. Convergence between biological, behavioural and genetic determinants of obesity. Nat Rev Genet. 2017;18(12):731–48. https://doi.org/10.1038/nrg.2017.72.
3. Iepsen EW, Zhang J, Thomsen HS, Hansen EL, Hollensted M, Madsbad S, et al. Patients with obesity caused by melanocortin-4 receptor mutations can be treated with a glucagon-like peptide-1 receptor agonist. Cell Metab. 2018;28(1):23–32 e3. https://doi.org/10.1016/j.cmet.2018.05.008.
4. •• Romere C, Duerrschmid C, Bournat J, Constable P, Jain M, Xia F, et al. Asprosin, a fasting-induced glucogenic protein hormone. Cell. 2016;165(3):566–79. https://doi.org/10.1016/j.cell.2016.02.063. This study discovered the hunger hormone asprosin for the first time.
5. Razani B, Combs TP, Wang XB, Frank PG, Park DS, Russell RG, et al. Caveolin-1-deficient mice are lean, resistant to diet-induced obesity, and show hypertriglyceridemia with adipocyte abnormalities. J Biol Chem. 2002;277(10):8635–47. https://doi.org/10.1074/jbc.M110970200.
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