ER stress-mediated cell damage contributes to the release of EDA+ fibronectin from hepatocytes in nonalcoholic fatty liver disease-Reference-Cited by-同舟云学术

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ER stress-mediated cell damage contributes to the release of EDA+ fibronectin from hepatocytes in nonalcoholic fatty liver disease

Published:2017-04 Issue:2 Volume:37 Page:217-225
ISSN:1672-0733
Container-title:Journal of Huazhong University of Science and Technology [Medical Sciences]
language:en
Short-container-title:J. Huazhong Univ. Sci. Technol. [Med. Sci.]

Author:

He Lei,Yuan Fa-hu,Chen Ting,Huang Qiang,Wang Yu,Liu Zhi-guo

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Biomedical Engineering,Biomaterials,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/s11596-017-1718-8.pdf

Reference37 articles.

1. Bedogni G, Miglioli L, Masutti F, et al. Prevalence of and risk factors for nonalcoholic fatty liver disease: The Dionysos Nutrition and Liver Study. Hepatology, 2005,42(1):44–52

2. Brunt EM. Pathology of nonalcoholic fatty liver disease. Nat Rev Gastroenterol Hepatol, 2010,7(4):195–203

3. Day CP, James OFW. Steatohepatitis: A tale of two “hits”? Gastroenterology, 1998,114(4):842–845

4. Ukkola O, Kesaniemi YA. Leptin and high-sensitivity C-reactive protein and their interaction in the metabolic syndrome in middle-aged subjects. Metabolism, 2007,56(9):1221–1227

5. Tan MH, Sun ZW, Opitz SL, et al. Deletion of the alternatively spliced fibronectin EIIIA domain in mice reduces atherosclerosis. Blood, 2004,104(1):11–18

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