Iron S Involvement in the Molecular Mechanisms and Pathogenesis of Alzheimers Disease-Reference-Cited by-同舟云学术

Iron S Involvement in the Molecular Mechanisms and Pathogenesis of Alzheimers Disease

Published:2003 Issue: Volume: Page:165-188
ISSN:
Container-title:Neuronal and Vascular Plasticity
language:
Short-container-title:

Author:

Henderson Rebecca J.,Connor James R.

Publisher

Springer US

Link

http://link.springer.com/content/pdf/10.1007/978-1-4615-0282-1_7.pdf

Reference58 articles.

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4. Bisaglia, M, V Venezia, P Piccioli, S Stanzione, C Porcile, C Russo, F Mancini, C Milanese, and G Schettini. (2002). Acetaminophen Protects Hippocampal Neurons and PC12 Cultures from Amyloid Beta-Peptides Induced Oxidative Stress and Reduces NF-kappaB Activation. Neurochemistry International. 41: 43–54.

5. Bruce, A, S Bose, W Fu, CM Butt, M-E Mirault, N Taniguchi, and MP Mattson. (1997). Amyloid Beta-Peptide Alters the Profile of Antioxidant Enzymes in Hippocampal Cultures in a Manner Similar to that Observed in Alzheimer’s Disease. Pathogenesis. 1: 15–30.

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1. Redox Metals in Alzheimer's Disease;Annals of the New York Academy of Sciences;2004-03