Glucose-6-Phosphate Dehydrogenase Deficiency and Primaquine Hemolytic Toxicity

Author:

Baird J. Kevin,Satyagraha Ari W.,Bancone Germana

Publisher

Springer New York

Reference38 articles.

1. Abeyaratne KP, Halpe NL. Sensitivity to primaquine in Ceylonese children due to deficiency of erythrocytic glucose-6-phophate dehydrogenase. Ceylon Med J. 1968;27:134–8.

2. Alving AS, Johnson CF, Tarlov AR, Brewer GJ, Kellermeyer RW, Carson PE. Mitigation of the hemolytic effect of primaquine and enhancement of its action against exoerythrocytic forms of Chesson strain of Plasmodium vivax by intermittent regimens of drug administration. A preliminary report. Bull WHO. 1960;22:621–9.

3. Anstey NM, Douglas NM, Poespoprodjo JR, Price RN. Plasmodium vivax: clinical spectrum, risk factors and pathogenesis. Adv Parasitol. 2012;80:151–201.

4. Baird JK. Evidence and implications of mortality associated with acute Plasmodium vivax malaria. Clin Microbiol Rev. 2013;26:36–57.

5. Baird JK, Hoffman SL. Primaquine therapy for malaria. Clin Infect Dis. 2004;37:1659–67.

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