The molecular biology of nasal polyposis-Reference-Cited by-同舟云学术

The molecular biology of nasal polyposis

Published:2001-05 Issue:3 Volume:1 Page:262-267
ISSN:1529-7322
Container-title:Current Allergy and Asthma Reports
language:en
Short-container-title:Curr Allergy Asthma Rep

Author:

Bernstein Joel M.

Publisher

Springer Science and Business Media LLC

Subject

Pulmonary and Respiratory Medicine,Immunology and Allergy,Immunology

Link

http://link.springer.com/content/pdf/10.1007/s11882-001-0017-3.pdf

Reference18 articles.

1. Khair OA, Davies RJ, Devalia JL: Bacteria-induced release of inflammatory mediators by bronchial epithelial cells. Eur Respir J 1996, 9:1913–1922.

2. Khair OA, Devalia JL, Abdelaziz MM, et al.: Effect of erythromycin on Haemophilus influenzae endotoxininduced release of IL-6, IL-8 and sICAM-1 by cultured human bronchial epithelial cells. Eur Respir J 1995, 9:1451–1457.

3. Khair OA, Devalia JL, Abdelaziz MM, et al.: Effect of Haemophilus influenzae endotoxin on the synthesis of IL-6, IL-8, TNF-alpha and expression of ICAM-1 in cultured human bronchial epithelial cells. Eur Respir J 1994, 12:2109–2116.

4. Opdahl H, Haugen T, Hagberg IA, et al.: Effects of short-term nitrogen monoxide inhalation on leukocyte adhesion molecules, generation of reactive oxygen species, and cytokine release in human blood. Nitric Oxide J 2000, 4:112–122. Increased nitrogen monoxide (NO) concentrations change leukocyte function under many experimental conditions. NO inhalation does not change the expression of cell adhesion molecules CD11a, CD11b, CD11c, and L-selectin on neutrophilic granulocytes or monocytes. However, the generation of reactive oxygen species in response to activation with phorbol myristate acetate increased in polymorphonuclear neutrophils after NO inhalation. Finally, baseline and lipopolysaccharide-stimulated production of IL-1b decreased after NO inhalation, whereas the lipopolysaccharide-stimulation production of TNF-a increased.

5. Krunkosky TM, Fischer VM, Martin LD, et al.: Effects of TNFalpha on expression of ICAM-1 in human airway epithelial cells in vitro: signaling pathways controlling surface and gene expression. Am J Respir Cell Mol Biol 2000, 22:685–692.

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