TLR9/FCRL3 regulates B cell viability, apoptosis, and antibody and IL-10 production through ERK1/2, p38, and STAT3 signaling pathways
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Developmental Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11626-022-00720-8.pdf
Reference27 articles.
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2. Calahorra L, Camacho-Toledano C, Serrano-Regal MP, Ortega MC, Clemente D (2022) Regulatory cells in multiple sclerosis: from blood to brain. Biomedicines 10(2):335. https://doi.org/10.3390/biomedicines10020335
3. Cavallo S (2020) Immune-mediated genesis of multiple sclerosis. J Transl Autoimmun 3:100039
4. Cencioni MT, Ali R, Nicholas R, Muraro PA (2021) Defective CD19+CD24hiCD38hi transitional B-cell function in patients with relapsing-remitting MS. Mult Scler 27(8):1187–1197
5. Cui X, Liu CM, Liu QB (2020) Fcrl3 promotes il-10 expression in b cells through the shp-1 and p38 mapk signaling pathways. Cell Biol Int 44(9):1811–1819
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1. Integrated analyses of single-cell transcriptome and Mendelian randomization reveal the protective role of FCRL3 in multiple sclerosis;Frontiers in Immunology;2024-07-15
2. Silencing of FCRLB by shRNA ameliorates MuSK-induced EAMG in mice;Journal of Neuroimmunology;2023-10
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