Genome-wide analysis of tunicamycin-induced endoplasmic reticulum stress response and the protective effect of endoplasmic reticulum inhibitors in neonatal rat cardiomyocytes-Reference-Cited by-同舟云学术

Genome-wide analysis of tunicamycin-induced endoplasmic reticulum stress response and the protective effect of endoplasmic reticulum inhibitors in neonatal rat cardiomyocytes

Published:2016-01-06 Issue:1-2 Volume:413 Page:57-67
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Liu Chun-Lei,Zhong Wu,He Yun-Yun,Li Xin,Li Song,He Kun-Lun

Funder

the Ministry Science Foundation of the Chinese People’s Liberation Army during the 12th Five-Year Plan Period

Major International Science and Technology Cooperation Projects

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11010-015-2639-0.pdf

Reference31 articles.

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2. Boyce M, Yuan J (2006) Cellular response to endoplasmic reticulum stress: a matter of life or death. Cell Death Differ 13(3):363–373

3. Minamino T, Komuro I, Kitakaze M (2010) Endoplasmic reticulum stress as a therapeutic target in cardiovascular disease. Circ Res 107(9):1071–1082

4. Deng Q, Xu C, Wang R, Zheng J, Li S, Jin Q, Yang Y (2015) TN-2 Ameliorates tunicamycin-induced mitochondria and endoplasmic reticulum stress-associated apoptosis in rat dorsal root ganglion neurons. J Mol Neurosci 57(2):314

5. Quan X, Wang J, Liang C, Zheng H, Zhang L (2015) Melatonin inhibits tunicamycin-induced endoplasmic reticulum stress and insulin resistance in skeletal muscle cells. Biochem Biophys Res Commun 463(4):1102–1107

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