Subchronic exposure to high-dose ACE-inhibitor moexipril induces catalase activity in rat liver-Reference-Cited by-同舟云学术

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Subchronic exposure to high-dose ACE-inhibitor moexipril induces catalase activity in rat liver

Published:2005-12 Issue:1-2 Volume:280 Page:159-163
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Adeghate E.,Hasan M. Y.,Ponery A. S.,Nurulain S. M.,Petroianu G. A.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11010-005-8843-6.pdf

Reference23 articles.

1. Gerstein HC: Reduction of cardiovascular events and microvascular complications in diabetes with ACE inhibitor treatment: HOPE and MICRO-HOPE. Diabetes Metab Res Rev 18: 82–85, 2002

2. Zanella MT, Kohlmann O Jr, Ribeiro AB: Treatment of obesity hypertension and diabetes syndrome. Hypertension 38: 705–708, 2001

3. McFarlane SI, Kumar A, Sowers JR: Mechanisms by which angiotensin-converting enzyme inhibitors prevent diabetes and cardiovascular disease. Am J Cardiol 91: 30–37, 2003

4. Mehendale HM: PPAR-alpha: A key to the mechanism of hepatoprotection by clofibrate. Toxicol Sci 57: 187–190, 2000

5. Sakamoto J, Kimura H, Moriyama S, Odaka H, Momose Y, Sugiyama Y, Sawada H: Activation of human peroxisome proliferator-activated receptor (PPAR) subtypes by pioglitazone. Biochem Biophys Res Commun 278: 704–711, 2000

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1. The current approach into signaling pathways in pulmonary arterial hypertension and their implication in novel therapeutic strategies;Pharmacological Reports;2014-08

2. Enalaprilat increases PPARβ/δ expression, without influence on PPARα and PPARγ, and modulate cardiac function in sub-acute model of daunorubicin-induced cardiomyopathy;European Journal of Pharmacology;2013-08

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