Restoration of Dystrophin Expression in Mdx-Derived Muscle Progenitor Cells Using CRISPR/Cas9 System and Homology-Directed Repair Technology
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Publisher
Springer US
Link
https://link.springer.com/content/pdf/10.1007/978-1-0716-2772-3_23
Reference8 articles.
1. Jelinkova S, Fojtik P, Kohutova A, Vilotic A, Marková L, Pesl M, Jurakova T, Kruta M, Vrbsky J, Gaillyova R et al (2019) Dystrophin deficiency leads to genomic instability in human pluripotent stem cells via NO synthase-induced oxidative stress. Cells 8:53
2. Jin Y, Shen Y, Su X, Weintraub N, Tang Y (2019) CRISPR/Cas9 technology in restoring dystrophin expression in iPSC-derived muscle progenitors. J Vis Exp
3. Jin Y, Shen Y, Su X, Cai J, Liu Y, Weintraub NL, Tang Y (2020a) The small GTPases Rab27b regulates mitochondrial fatty acid oxidative metabolism of cardiac mesenchymal stem cells. Front Cell Dev Biol 8:209–209
4. Jin Y, Shen Y, Su X, Weintraub NL, Tang Y (2020b) Effective restoration of dystrophin expression in iPSC (Mdx)-derived muscle progenitor cells using the CRISPR/Cas9 system and homology-directed repair technology. Comput Struct Biotechnol J 18:765–773
5. Jin Y, Shen Y, Weintraub NL, Tang Y (2020c) Using iRFP genetic labeling technology to track tumorogenesis of transplanted CRISPR/Cas9-edited iPSC in skeletal muscle. Methods Mol Biol 2126:73–83
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