Alcohol Intake Provoked Cardiomyocyte Apoptosis Via Activating Calcium-Sensing Receptor and Increasing Endoplasmic Reticulum Stress and Cytosolic [Ca2+]i-Reference-Cited by-同舟云学术

Alcohol Intake Provoked Cardiomyocyte Apoptosis Via Activating Calcium-Sensing Receptor and Increasing Endoplasmic Reticulum Stress and Cytosolic [Ca2+]i

Published:2023-08-28 Issue:4 Volume:81 Page:707-716
ISSN:1085-9195
Container-title:Cell Biochemistry and Biophysics
language:en
Short-container-title:Cell Biochem Biophys

Author:

Liu Wenxiu,Zhao Meng,Zhang Xin,Chi Jinyu,Yin Xinhua,Liu Yue

Funder

National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Biochemistry,General Medicine,Biophysics

Link

https://link.springer.com/content/pdf/10.1007/s12013-023-01167-8.pdf

Reference43 articles.

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2. Movva, R., & Figueredo, V. M. (2013). Alcohol and the heart: to abstain or not to abstain? International Journal of Cardiology, 164(3), 267–276.

3. Regula, K. M., & Kirshembaum, L. A. (2005). Apoptosis of ventricular myocytes; a means to an end. Journal of Molecular and Cellular Cardiology, 38(1), 3–13.

4. Thomas, A. P., Sass, E. J., & Tun-Kirchmann, T. T., et al. (1989). Ethanolinhibits electrically-induced calcium transients in isolated rat cardiac myocytes. Journal of Molecular Cellular Cardiology, 21(6), 555–565.

5. Li, S. Y., & Ren, J. (2008). Cardiac overexpression of alcohol dehydrogenase exacerbates chronic ethanol ingestion-induced myocardial dysfunction and hypertrophy: role of insulin signaling and ER stress. Journal of Molecular and Cellular Cardiology, 44(6), 992–1001.

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