Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice-Reference-Cited by-同舟云学术

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Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice

Published:2015-03-03 Issue:5 Volume:35 Page:713-721
ISSN:0272-4340
Container-title:Cellular and Molecular Neurobiology
language:en
Short-container-title:Cell Mol Neurobiol

Author:

Lu Xin-Yu,Wang Han-Dong,Xu Jian-Guo,Ding Ke,Li Tao

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Cellular and Molecular Neuroscience,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s10571-015-0167-9.pdf

Reference47 articles.

1. Anrather J, Racchumi G, Iadecola C (2006) NF-kappaB regulates phagocytic NADPH oxidase by inducing the expression of gp91phox. J Biol Chem 281:5657–5667

2. Beziaud T, Ru CX, El SN, Frechou M, Teng F, Palmier B, Beray-Berthat V, Soustrat M, Margaill I, Plotkine M, Marchand-Leroux C, Besson VC (2011) Simvastatin in traumatic brain injury: effect on brain edema mechanisms. Crit Care Med 39:2300–2307

3. Cairns B, Kim JY, Tang XN, Yenari MA (2012) NOX inhibitors as a therapeutic strategy for stroke and neurodegenerative disease. Curr Drug Targets 13:199–206

4. Chen H, Song YS, Chan PH (2009) Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion. J Cereb Blood Flow Metab 29:1262–1272

5. Cheng ZG, Zhang GD, Shi PQ, Du BS (2013) Expression and antioxidation of Nrf2/ARE pathway in traumatic brain injury. Asian Pac J Trop Med 6:305–310

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2. Neurological Manifestations Following Traumatic Brain Injury: Role of Behavioral, Neuroinflammation, Excitotoxicity, Nrf-2 and Nitric Oxide;CNS & Neurological Disorders - Drug Targets;2024-07-30

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4. 20(R)‐ginsenoside Rg3 attenuates cerebral ischemia–reperfusion injury by mitigating mitochondrial oxidative stress via the Nrf2/HO‐1 signaling pathway;Phytotherapy Research;2024-01-21

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