Progeria: Model Organisms
Author:
Publisher
Springer International Publishing
Link
https://link.springer.com/content/pdf/10.1007/978-3-030-22009-9_723
Reference47 articles.
1. Arancio W, Pizzolanti G, Genovese SI, Pitrone M, Giordano C (2014) Epigenetic involvement in Hutchinson-Gilford progeria syndrome: a mini-review. Gerontology 60(3):197–203. https://doi.org/10.1159/000357206
2. Baek JH, Schmidt E, Viceconte N, Strandgren C, Pernold K, Richard TJ, Van Leeuwen FW, Dantuma NP, Damberg P, Hultenby K, Ulfhake B, Mugnaini E, Rozell B, Eriksson M (2015) Expression of progerin in aging mouse brains reveals structural nuclear abnormalities without detectible significant alterations in gene expression, hippocampal stem cells or behavior. Hum Mol Genet 24(5):1305–1321
3. Bar DZ, Gruenbaum Y (2010) Reversal of age-dependent nuclear morphology by inhibition of prenylation does not affect lifespan in Caenorhabditis elegans. Nucleus 1(6):499–505. https://doi.org/10.4161/nucl.1.6.13223
4. Bar DZ, Neufeld E, Feinstein N, Gruenbaum Y (2009) Gliotoxin reverses age-dependent nuclear morphology phenotypes, ameliorates motility, but fails to affect lifespan of adult Caenorhabditis elegans. Cell Motil Cytoskeleton 66:791–797
5. Beard GS, Bridger JM, Kill IR, Tree DR (2008) Towards a Drosophila model of Hutchinson-Gilford progeria syndrome. Biochem Soc Trans 36(Pt 6):1389–1392. https://doi.org/10.1042/BST0361389
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