Cellular clones and transgenic mice overexpressing copper — zinc superoxide dismutase: Models for the study of free radical metabolism and aging

Author:

Ceballos-Picot Irène,Nicole Annie,Sinet Pierre-Marie

Publisher

Birkhäuser Basel

Reference46 articles.

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2. Anneren, G., and Epstein, C. J. (1987) Lipid peroxidation and superoxide dismutase-1 and glutathione peroxidase activities in trisomy 16 fetal mice and human trisomy 21 fibroblasts. Pediat. Res. 21: 88–92.

3. Auh, B., Caviedes, P., Hidalgo, J., Epstein, C. J., and Rapoport, S. L (1989) Electrophysiological analysis of cultured fetal mouse dorsal root ganglion neurons transgenic for human superoxide dismutase-gene in the Down syndrome region of chromosome 21. Brain Res. 497:191 -194.

4. Avraham, K. B., Schickler, M., Sapoznokow, D., Yarom, R., and Groner, Y. (1988) Down’s syndrome: abnormal neuromuscular junction in tongue of transgenic mice with elevated levels of human CuZn-superoxide dismutase. Cell 54: 823–829.

5. Brooksbank, B. W. L., and Balazs, R. (1984) Superoxide dismutase, glutathione peroxidase and lipoperoxidation in Down syndrome fetal brain. Dev. Brain Res. 16: 37–44.

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