Consequences of Neurotoxin-Induced Dopamine Loss on Striatal Synaptic Plasticity

Author:

Gibson Anne S.,Keefe Kristen A.

Publisher

Springer International Publishing

Reference50 articles.

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2. Aragona, B. J., Cleaveland, N. A., Stuber, G. D., Day, J. J., Carelli, R. M., & Wightman, R. M. (2008). Preferential enhancement of dopamine transmission within the nucleus accumbens shell by cocaine is attributable to a direct increase in phasic dopamine release events. The Journal of Neuroscience, 28(35), 8821–8831. https://doi.org/10.1523/JNEUROSCI.2225-08.2008

3. Barker-Haliski, M. L., Oldenburger, K., & Keefe, K. A. (2012). Disruption of subcellular Arc/Arg 3.1 mRNA expression in striatal efferent neurons following partial monoamine loss induced by methamphetamine. Journal of Neurochemistry, 123(5), 845–855. https://doi.org/10.1111/jnc.12017

4. Bergstrom, B. P., & Garris, P. A. (2003). “Passive stabilization” of striatal extracellular dopamine across the lesion spectrum encompassing the presymptomatic phase of Parkinson’s disease: A voltammetric study in the 6-OHDA-lesioned rat. Journal of Neurochemistry, 87(5), 1224–1236. https://doi.org/10.1046/j.1471-4159.2003.02104.x

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