Extracellular signal-regulated kinase is activated in podocytes from patients with diabetic nephropathy
Author:
Funder
National Defense Medical Colledge
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s13577-024-01108-4.pdf
Reference30 articles.
1. Gross JL, de Azevedo MJ, Silveiro SP, LsH Canani, Caramori ML, Zelmanovitz T. Diabetic nephropathy: diagnosis, prevention, and treatment. Diabetes Care. 2005;28(1):164–76. https://doi.org/10.2337/diacare.28.1.164.
2. Barutta F, Bellini S, Gruden G. Mechanisms of podocyte injury and implications for diabetic nephropathy. Clin Sci. 2022;136(7):493–520. https://doi.org/10.1042/cs20210625.
3. Hoshi S, Nomoto K, Kuromitsu J, Tomari S, Nagata M. High glucose induced VEGF expression via PKC and ERK in glomerular podocytes. Biochem Biophys Res Commun. 2002;290(1):177–84. https://doi.org/10.1006/bbrc.2001.6138.
4. Kriz W, Gretz N, Lemley KV. Progression of glomerular diseases: is the podocyte the culprit? Kidney Int. 1998;54(3):687–97. https://doi.org/10.1046/j.1523-1755.1998.00044.x.
5. Nagata M, Nakayama K, Terada Y, Hoshi S, Watanabe T. Cell cycle regulation and differentiation in the human podocyte lineage. Am J Pathol. 1998;153(5):1511–20. https://doi.org/10.1016/s0002-9440(10)65739-2.
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