MEK/ERK Signaling in β-Cells Bifunctionally Regulates β-Cell Mass and Glucose-Stimulated Insulin Secretion Response to Maintain Glucose Homeostasis-Reference-Cited by-同舟云学术

MEK/ERK Signaling in β-Cells Bifunctionally Regulates β-Cell Mass and Glucose-Stimulated Insulin Secretion Response to Maintain Glucose Homeostasis

Published:2021-04-27 Issue:7 Volume:70 Page:1519-1535
ISSN:0012-1797
Container-title:Diabetes
language:en
Short-container-title:Diabetes

Author:

Ikushima Yoshiko Matsumoto,Awazawa Motoharu,Kobayashi Naoki,Osonoi Sho,Takemiya Seiichi,Kobayashi Hiroshi,Suwanai Hirotsugu,Morimoto Yuichi,Soeda Kotaro,Adachi Jun,Muratani Masafumi,Charron Jean,Mizukami Hiroki,Takahashi Noriko,Ueki Kohjiro^ORCID

Publisher

American Diabetes Association

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Reference51 articles.

1. Islet β cell mass in diabetes and how it relates to function, birth, and death

2. Mechanisms linking obesity to insulin resistance and type 2 diabetes

3. The ERK Cascade: Distinct Functions within Various Subcellular Organelles

4. The Regulatory Roles of Mitogen-Activated Protein Kinase (MAPK) Pathways in Health and Diabetes: Lessons Learned from the Pancreatic β-Cell

5. Functional redundancy of the kinases MEK1 and MEK2: Rescue of the Mek1 mutant phenotype by Mek2 knock-in reveals a protein threshold effect

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